为探究 AD 神经发生性别差异机制,研究人员聚焦微胶质细胞 Atg5,发现其有性别特异性作用,为 AD 治疗提供新思路。
The scientists found this mechanism by using advanced techniques such as PET imaging, multi-omics and 3D kinematics and investigating mice lacking ATG5. As a result, GLUT2 accumulated, which led ...