Here, we describe roles of miR160-directed ARF17 posttranscriptional regulation. Plants expressing a miRNA-resistant version of ARF17 have increased ARF17 mRNA levels and altered accumulation of auxin-inducible GH3-like mRNAs, YDK1/GH3.2, GH3.3, GH3.5, and DFL1/GH3.6, which encode auxin-conjugating proteins.

2020年6月10日 · In our study, the overexpression of miR160a in the transgenic lines resulted in inhibition of the target genes ARF10, ARF16, ARF17, as well as GH3.5-like mRNA, resulting in elevated levels of active IAA and increased biomass of …

2006年1月1日 · As a principal component of the RNA-induced silencing complex, AGO1 protein has a pleiotropic effect on the processing of microRNA cleavage of the target mRNAs. It was reported that ARF17 was over-accumulated and the expression of several auxin-inducible GH3 genes were down-regulated in hypocotyls of ago1 mutant Arabidopsis ( 28).

2019年9月14日 · Catherine Bellini研究组在之前的研究中发现，ARF6 和 ARF8是拟南芥不定根形成的正调控因子，其通过促进GH3 (Gretchen Hagen 3) 基因的表达，负调控JA的积累，从而促进不定根的发生。

2005年4月29日 · Plants expressing a miRNA-resistant version of ARF17 have increased ARF17 mRNA levels and altered accumulation of auxin-inducible GH3-like mRNAs, YDK1/GH3.2, GH3.3, GH3.5, and DFL1/GH3.6, which encode auxin-conjugating proteins. These expression changes correlate with dramatic developmental defects, including embryo and emerging leaf symmetry ...

Here, we describe roles of miR160-directed ARF17 posttranscriptional regulation. Plants expressing a miRNA-resistant version of ARF17 have increased ARF17 mRNA levels and altered accumulation of auxin-inducible GH3 -like mRNAs, YDK1/GH3.2, GH3.3, GH3.5, and DFL1/GH3.6, which encode auxin-conjugating proteins.

that disrupting miR160 regulation of ARF17 increases ARF17 mRNA levels, leads to severe developmental abnormalities, including defects in embryonic, root, vegetative, and floral de-velopment, and alters GH3-like gene expression. These results indicate that miR160-directed regulation is critical for the de-

The authors suggest a model in which ARF17 negatively regulates GH3 gene expression and adventitious root formation in Arabidopsis, through its regulation by AGO1-dependent microRNA-mediated repression.

ARF6, ARF8 and ARF17 act upstream of GH3.3, GH3.5 and GH3.6 to stimulate AR development by modulating jasmonate homeostasis, demonstrating that jasmonate homeostasis is regulated by auxin in Arabidopsis (Gutierrez et al., 2012).

2021年2月1日 · Here we show that miR160 and its target genes ARF10, ARF16 and ARF17 modulate hypocotyl elongation in a light, brassinazole (BRZ, a BR biosynthesis inhibitor), or paclobutrazol (PAC, a GA biosynthesis inhibitor)-dependent manner in Arabidopsis. miR160, ARF10, ARF16 and ARF17 have no effects on hypocotyl elongation in the dark.