We report outcome of treatment with local intra-arterial thrombolysis administered 2.75 hours after devastating visual loss from incomplete CRAO. Keywords: retina, eye. Central retinal artery occlusion (CRAO) is a devastating ocular condition that results in …

Twenty-eight well-documented cases of NVI after CRAO were detected, an occurrence rate of 16.6%. After evaluating the intravenous fluorescein an giograms of these patients, the authors conclude that an extreme prolongation of the retinal arteriovenous transit time is an independent risk factor for the subsequent development of NVI.

Acute nonarteritic central retinal artery occlusion (CRAO) is an eye stroke with poor visual prognosis and no proven effective therapies. Given advances in acute stroke care, thrombolysis in CRAO merits critical re-examination. We review the evidence for intravenous (IV) and intra-arterial (IA) tissue plasminogen activator (tPA) in CRAO management.

Numerous therapeutic options have been suggested for the treatment of central retinal artery occlusion (CRAO) such as ocular massage, anterior chamber paracentesis, physical exercise, and medication-induced reduction of intraocular pressure.

There are a number of therapies that has been used in the treatment of CRAO in the past. These include carbogen inhalation, acetazolamide infusion, ocular massage and paracentesis, as well as various vasodilators such as intravenous glyceryl trinitrate.

2018年7月1日 · Intravenous fluorescein angiography (IVFA), optical coherence tomography (OCT), and OCT angiography may be used in diagnosis by identifying areas of delayed or absent retinal perfusion. 29 Intravenous FA can also help to group patients into CRAO subtypes (complete, incomplete, cilioretinal sparing).

Central retinal artery occlusion (CRAO) is an ophthalmic emergency and the ocular analogue of cerebral stroke. Best evidence reflects that over three-quarters of patients suffer profound acute visual loss with a visual acuity of 20/400 or worse.

Three types of manifestations were identified by FFA in 63 eyes, including poor perfusion (18 cases), exudation (22 cases) and mixed types (23 cases) of CRAO. No significant difference was found in age (F=0.171, P=0.844) and disease course (F=0.016, P=0.984) among the three types of …

CRAO: Harbinger of Ischemic Stroke RETINA CLINICAL UPDATE Marc H. Levin, MD, PhD C ontroversy persists about how best to manage patients with acute central retinal artery oc-clusion (CRAO) and how much can be done to recover their vision—especially given how quickly the retina experi-ences irreversible ischemia following a CRAO.

Central retinal artery occlusion (CRAO) is caused by partial or complete occlusion of the central retinal artery, most commonly by an embolus from the ipsilateral carotid artery, aortic arch, or heart, and is the ocular equivalent of an acute cerebral ischemic event.