2023年9月1日 · Flair vessel hyperintensity (FVH) is caused by blood flow with reduced velocity. FVH indicates slow anterograde or collateral flow in large artery steno-occlusion. FVH is associated with impaired perfusion and penumbra in ischemic stroke. Absence of FVH in large artery occlusion means either robust or poor collateral flow.

2022年5月1日 · Fluid-attenuation inversion recovery (FLAIR) vascular hyperintensity (FVH) is a common presentation on brain magnetic resonance images of patients with acute ischemic stroke. This sign is known as a sluggish collateral flow.

2011年11月1日 · As opposed to other vessel signs seen in arterial insufficiency, FVH is unique in that it does not represent thrombus, but rather sluggish or disordered blood flow through vessels. This review will discuss the diagnostic and prognostic value of FVH and its impact on clinical decision-making.

2016年1月5日 · Fluid-attenuated inversion recovery vascular hyperintensities (FVH) beyond the boundaries of diffusion-weighted imaging (DWI) lesion (FVH-DWI mismatch) have been proposed as an alternative to perfusion-weighted imaging (PWI)-DWI mismatch.

2013年5月7日 · High signal intensity within blood vessels on fluid-attenuated inversion recovery (FLAIR), otherwise known as FLAIR vascular hyperintensity (FVH), is often observed in patients with acute ischemic stroke (AIS) attributable to large artery stenosis or occlusion. 1–9 Altered hemodynamics, such as stationary and slow blood flow often from collatera...

2019年2月14日 · Fluid-attenuated inversion recovery vascular hyperintensity (FVH) is frequently found in stroke patients after intracranial arterial occlusion, but the prognostic value of FVH findings is unclear.

Fluid-attenuated inversion recovery (FLAIR) vascular hyperintensity (FVH) is frequently encountered in patients with acute ischemic stroke and significant intracranial arterial stenosis or occlusion. The mechanisms underlying this phenomenon and the clinical implications of FVH have been a matter of debate.

2024年11月8日 · Background: Fluid-attenuated inversion recovery (FLAIR) vascular hyperintensity (FVH) might be useful for predicting and functional outcome in ischemic stroke patients after endovascular thrombectomy (EVT), but its clinical benefit remains controversial.

Through reviewing the available studies on the etiologic mechanism of FVH and investigating findings on its clinical significance in AIS and TIA, this review aims to elucidate the key factors for interpreting the clinical significance of FVH individually.

The presence, location, and morphology of FVH were noted and correlated with markers of thrombotic occlusion and collateral flow on angiography. Angiographic collaterals were graded on a 5-point scale incorporating extent and hemodynamic aspects.