2017年1月8日 · The key parameter I want to emphasize is the functional residual capacity (FRC) – the air remaining in our lungs after a passive expiration. At FRC, the inward elastic forces of the lung are in equilibrium with the outward elastic forces of the chest wall. With this definition in mind, pathologies which change these elastic forces will also ...

2024年1月31日 · Pulmonary vascular resistance (PVR) is the resistance against blood flow from the 4 pulmonary veins of the lung to the left atrium. It is most commonly modeled using a modification of Ohm’s law (see Image.

2022年12月9日 · 功能残气量（functional residual capacity, FRC）是平静呼气后肺内所含有的气体总量。 在此状态下，肺内压与大气压保持平衡，胸内压=肺弹性回缩压，肺组织向内的弹性回缩力与胸廓向外的弹性扩张力达到平衡，此时吸气阻力最小、肺血管阻力（pulmonary vascular resistance, PVR）最低、循环状态最稳定。 由图1可知：FRC=ERV+RV，由于RV（residual volume）不能通过肺量计直接测得，故临床上FRC也不能用肺量计直接测出，其测定需要借 …

2019年11月14日 · In short, the relationship between lung volume and PVR can be summarised into three pointform statements and expressed via a helpfully memorable and highly textbook-represented diagram: Pulmonary vascular resistance is lowest at FRC; At low lung volumes, it increases due to the compression of larger vessels

肺功能评估指标傻傻分不清？ 看这儿一篇文章教你搞懂 FRC, RV, ERV, IC, VC 这些肺功能指标! 刚开始接触心肺康复的同学们，可能看到这个图都觉得晕，那么多指标，下面三大表格教你搞清楚~ 肺容积 (lung volumes)指标. 肺容量 ( lung capacities)指标. 肺通气功能评估 指标 ...

2024年12月31日 · 在健康状态下，假定所有肺单位通畅，这些相反的通气效应通常在功能残气容量（frc）时最优化。 肺通气对血管阻力的影响及其与肺容积之间的关系。 原文下载 下载

Hypoxic pulmonary vasoconstriction (HPV) represents an intrinsic property of pulmonary vascular smooth muscle cells (VSCMs) and an important homeostatic mechanism that helps to ensure correct gas exchange in physiological and pathological conditions.

2018年1月8日 · Alveolar and extra-alveolar PVRs are added together to result in a cumulative pulmonary vascular PVR at a specific lung volume, which is low at the functional residual capacity (FRC) and high at both residual volume (RV) and total lung capacity (TLC) thereby resulting in a U shaped cumulative PVR curve . Mechanical ventilation with positive end ...

Pulmonary vascular resistance (PVR), the major determinant of RV afterload, is minimal near FRC and related to lung volume in a bimodal fashion. Atelectatic alveoli compress extra–alveolar vessels while alveolar overdistension compresses intra–alveolar vessels, both resulting in an increase in PVR.

Rationale: A U-shaped relationship should exist between lung volume and pulmonary vascular resistance (PVR), with minimal PVR at FRC. Thus, positive end-expiratory pressure (PEEP) in patients with acute respiratory distress syndrome (ARDS) should increase PVR if it induces significant lung distension compared with recruitment.