
Therapeutic targeting of VEGFR2 in HBV-associated hepatocellular ...
2021年5月7日 · In The Lancet Gastroenterology & Hepatology, Shukui Qin and colleagues 6 report findings of the AHELP trial, in which apatinib, a selective inhibitor of vascular endothelial …
Expression and prognostic value of VEGFR-2, PDGFR-β, and c …
2013年4月3日 · We found that in patients with HCC, high expression of VEGFR-2 correlates with chronic hepatitis B virus infection and hepatic cirrhosis. High expression of PDGFR-β is a …
VEGF Upregulation in Viral Infections and Its Possible Therapeutic ...
VEGF expression was found to be upregulated along with COX-2 in tissue sections from human HCC with HBV infection and this expression was found to be positively correlated with …
Hepatitis B x antigen up-regulates vascular endothelial growth …
2007年5月30日 · The results identified elevated vascular endothelial growth factor receptor-3 short form splice variant (VEGFR-3 S) expression in HBxAg positive compared to negative …
Therapeutic targeting of VEGFR2 in HBV-associated hepatocellular ...
Pinato, D. J., Valeri, N., Muhammed, A., & Cortellini, A. (2021). Therapeutic targeting of VEGFR2 in HBV-associated hepatocellular carcinoma. The Lancet Gastroenterology and Hepatology, 6 …
CXCL5/CXCR2/VEGF通路在HBV相关肝癌血管生成中的作用及其机制
用CCK-8法、ELISA、血管生成实 验、qPCR 法、WB 法和免疫荧光染色技术分别检测过表达 CXCL5 对 HepG2 细胞增殖能力、VEGF 分泌、成管能力、CXCL5、 CXCR2、VEGF mRNA …
索拉非尼治疗 HBV 相关性肝癌过程中患者血清VEGFR-2及 AFP 水 …
方法选取符合巴塞罗那诊断标准的中晚期HBV相关性肝癌患者46例,随机分为2组,每组23例,对照组给予抗HBV、对症、支持等常规治疗,治疗组在常规治疗的基础上加用索拉非尼400 mg …
慢性HBV感染者Th17/Treg细胞因子、VEGF、GP73表达水平差异与 …
探讨慢性乙肝病毒(hepatitis B virus,HBV)感染者外周血Th17/Treg细胞因子[白细胞介素(IL)-6、IL-17、IL-10和转化生长因子-β(TGF-β)]及高尔基体糖蛋白-73(Golgi protein …
Ki67、CD34、VEGF在HBV和HCV相关性肝癌中表达的对比研究及 …
肝炎病毒 (hepatitis virus)的感染是导致HCC的主要因素,在我国以乙型肝炎病毒 (hepatitis B virus, HBV)为主,但随着乙肝疫苗的普及应用及卫生条件的改善,HBV感染率持续下降,HBV感染相关 …
Levels of HBx, VEGF, and CEACAM1 in HBV-related hepatocellular …
HBx, VEGF and CEACAM1 were widely expressed in HBV-related HCC. HBx may facilitate occurrence and progression of HBV-related HCC via down-regulating CEACAM1 and up …
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